![]() In addition to requiring the Blood Magic status, each Blood Magic spell also has a Blood Cost to cast. Once used, you will have access to Blood Magic for the following 2 turns: Feedback is welcome on whether this is too low or too high, but it is intended to make it a tactical choice to utilise your Blood Magic at the expense of vitality, rather than just having overpowered abilities from the offset. At the moment, the damage is set to be a total of 35% of your maximum health, ignoring armor. When activated, you will wound yourself, empowering you to use your powerful Blood Magic abilities. In addition to the above, you will start with the ability "Blood Magic" which costs no memory slots and no action points. This status is essentially a "Bleeding" status that is not affected by armor.īlood Spears: Shoot sharp magical spears of blood at up to 3 enemies, dealing physical damage and setting Lacerated.īlood Explosion: Throw an orb of blood at an enemy, which will explode on impact dealing physical damage and creating a cursed blood surface.īlood Jump: Magically propel youself to a target location, creating a blood surface and setting Lacerated on enemies in the impact zone. The skills also often either inflict the status Lacerated, or otherwise only effect targets already afflicted with Lacerated. These are also not really any more or less powerful than vanilla skills, just different. ![]() The class has some basic bloody spells, which are memorised as usual and take memory slots. Therefore, to use your most powereful Blood spells, you must sacrifice some of your own vitality. Whereas vanilla Necros gain health through damaging enemies, the idea behind the Blood Mage is to use your own life force to empower your skills. The Blood Mage is different to the vanilla Necromancer, and so I've made the skills fit the "Special" category, rather than Necromancer. This is a class I really wanted to make for myself, and I feel like it's turned out more or less how I wanted. So, I've loved the idea of a Blood Mage since Dragon Age:Origins.
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It is the most common cause of bacterial conjunctivitis.įIGURE 15–6 Coagulase test-Upper tube inoculated with Staphylococcus aureus lower tube inoculated with Staphylococcus epidermidis. It is an important cause of skin infections, such as folliculitis ( Figure 15–3), cellulitis, and impetigo ( Figure 15–4). It is one of the most common causes of hospital-acquired pneumonia, septicemia, and surgical-wound infections. Staphylococcus aureus causes abscesses ( Figure 15–1), various pyogenic infections (e.g., endocarditis, septic arthritis, and osteomyelitis), food poisoning, scalded skin syndrome ( Figure 15–2), and toxic shock syndrome. (2) Biochemically, staphylococci produce catalase (i.e., they degrade hydrogen peroxide), whereas streptococci do not. (1) Microscopically, staphylococci appear in grapelike clusters, whereas streptococci are in chains. Staphylococci and streptococci are nonmotile and do not form spores.īoth staphylococci and streptococci are gram-positive cocci, but they are distinguished by two main criteria: Two of the most important human pathogens, Staphylococcus aureus and Streptococcus pyogenes, are described in this chapter. As a result of high rates of resistance in currently recommended therapy and prophylaxis, the choice of optimal antibiotic therapy is vital in the individual patient.There are two medically important genera of gram-positive cocci: Staphylococcus and Streptococcus. GPC were the most frequent bacteria in culture-positive SBP and a variety of drug-resistant microorganisms have emerged. Third-generation cephalosporin resistance was observed in 49% and quinolone resistance in 47%. More infections with GNB than GPC were healthcare-associated (81% vs 42%, P = 0.007). MDR bacteria were more frequently isolated in healthcare-associated than in community-acquired infections (100% vs 50%, P = 0.006), in patients receiving long-term quinolone prophylaxis (67% vs 24%, P = 0.013) and in those with advanced liver disease as suggested by higher MELD score (28 vs 19, P = 0.012). coli ( n = 3) and Pseudomonas aeruginosa ( n = 2). pneumonia ( n = 4), followed by extended-spectrum beta-lactamase-producing E. Nine of the isolated bacteria (19%) were MDR, including carbapenemase-producing K. The most prevalent organisms in a descending order were Streptococcus spp ( n = 10), Enterococcus spp ( n = 9), Escherichia coli ( n = 8), Klebsiella pneumonia ( n = 5), methicillin-sensitive Staphylococcus aureus ( n = 4) and coagulase-negative Staphylococcus spp ( n = 3). GPC were found to be the most frequent cause (55%). Twenty-eight (60%) patients had healthcare-associated infections while 15 (32%) received prophylactic quinolone treatment. We retrospectively recorded 47 cases (66% males) during a 4-year-period (2008–2011). To assess possible recent changes of the bacteria causing SBP in cirrhotic patients. Recently, an increasing rate of infections with Gram-positive cocci (GPC) and multidrug-resistant (MDR) microorganisms was demonstrated. Spontaneous bacterial peritonitis (SBP) is historically caused by Gram-negative bacteria (GNB) almost exclusively Enterobacteriaceae. |
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